Moga dapat bertahan

Hampir setiap hari berulang alik ke hospital,kerap nak pitam.Tapi masih lagi mengelak dari masuk wad.Selepas dimasukkan drip dan bila rasa bertenaga sedikit,minta discharge..Berkali2 macam ni,Sehinggakan doktor kata merbahaya kalau dibiarkan.Nasihat doktor,pentingkan kesihatan diri dulu..Nak sangat teruskan rawatan tapi masa dan keadaan belum mengizinkan.Cuba untuk bertahan selagi boleh.

Anemia masih dalam investigation..

Rawatan lanjut belum dapat diteruskan,selain Guilain Barre Syndrome,ada lagi penyakit yang belum dapat dikenal pasti.Sehingga kini belum dapat diketahui apa punca anemia.Jantung tak stabil,doctor bimbang takut serangan jantung.Anemia,sama ada disebabkan masalah usus,atau bone marrow failure atau sebagainya..Saya perlu menjalani colonoscopy,endoscopy,barrium, untuk mengetahui apakah masalah usus ni.Kian hari makin kronik.Sebelum ni buat endoscopy,dapat sahkan chronic gastritis.Result colonoscopy sebelum ni,usus terlalu nipis..Perlu repeat lagi.

Bagaimanala untuk menghadapi segala ujian ni?Fikiran ni sentiasa risau memikirkan kesihatan,tapi cuba diselindungkan.Gelak tawa anak2,senyuman mengubat hati..Kena juga kuatkan diri demi anak2.

Kesakitan yang ditanggung ini menjadi satu bebanan pada insan lain.Menjadi satu kebencian..Tiada siapa yang pinta untuk menanggung kesakitan.Ini ujian untuk diri ini...tabahlah wahai hati...

Irritable BoweL Syndrome

Perlu repeat semula endoscopy dan colonoscopy...masalah usus yang makin kronik. Masih lagi tidak tahu punca..

Irritable bowel syndrome (IBS or spastic colon) is a diagnosis of exclusion. It is a functional bowel disorder characterized by chronicabdominal pain, discomfort, bloating, and alteration of bowel habits in the absence of any detectable organic cause.^[1] In some cases, the symptoms are relieved by bowel movements.^[2] Diarrhea or constipation may predominate, or they may alternate (classified as IBS-D, IBS-Cor IBS-A, respectively). IBS may begin after an infection (post-infectious, IBS-PI), a stressful life event, or onset of maturity without any other medical indicators.

Although there is no cure for IBS, there are treatments that attempt to relieve symptoms, including dietary adjustments, medication andpsychological interventions. Patient education and a good doctor-patient relationship are also important.^[2]

Several conditions may present as IBS including coeliac disease, fructose malabsorption,^[3] mild infections, parasitic infections likegiardiasis,^[4] several inflammatory bowel diseases, functional chronic constipation, and chronic functional abdominal pain. In IBS, routine clinical tests yield no abnormalities, although the bowels may be more sensitive to certain stimuli, such as balloon insufflation testing. The exact cause of IBS is unknown. The most common theory is that IBS is a disorder of the interaction between the brain and thegastrointestinal tract, although there may also be abnormalities in the gut flora or the immune system.^[5][6]

IBS does not lead to more serious conditions in most patients.^{[7][8][9][10][11]} However, it is a source of chronic pain, fatigue, and other symptoms and contributes to work absenteeism.^[12][13] Researchers have reported that the high prevalence of IBS,^[14][15][16] in conjunction with increased costs, produces a disease with a high societal cost.^[17] It is also regarded as a chronic illness and can dramatically affect the quality of a sufferer's life.

Ecg Test

Semalam buat ecg test,result jantung tak comel...Mungkin sebab kekurangan darah merah.Hemoglobin makin menurun.Anemia..Tak boleh banyak bergerak sekarang ni,boleh pitam..Tapi gagahkan diri juga untuk anak2..harap2 dapat bertahan.Bertahan dan bertahan..

Indahnya hidup kalau tanpa sakit dan derita tapi walau betapa pahit dan getirnya....redha dan terima segala dugaan.

What is Bone Marrow Failure

The bone marrow failure syndromes include a group of disorders than can be either inherited or acquired. These diseases are disorders of the hematopoietic stem cell that can involve either one cell line or all of the cell lines (erythroid for red cells, myeloid for white blood cells, megakaryocytic for platelets). The lymphocytes, which are involved in lymphoproliferative disorders, are usually spared. The inherited bone marrow failure syndromes include Fanconi anemia, dyskeratosis congenital, Diamond-Blackfan anemia, and other genetic disorders. The most common cause of acquired bone marrow failure is aplastic anemia.¹ Other diseases that can present in a manner similar to acquired bone marrow failure includemyelodysplastic syndromes, paroxysmal nocturnal hemoglobinuria, and large granular lymphocyte leukemia.

Bone marrow failure can be inherited or acquired. It can involve just 1 cell line or all 3 cell lines. The pathophysiology of these defects includes the following mechanisms of action: (1) a decrease in or damage to the hematopoietic stem cells and their microenvironment, resulting in hypoplastic or aplastic bone marrow; (2) maturation defects, such as vitamin B-12 or folate deficiency; and (3) differentiation defects, such as myelodysplasia.

Generally, hematopoietic stem cells are damaged by a congenital defect or exposure to a noxious substance or factor. Pathophysiologic mechanisms are (1) an acquired stem cell injury from viruses, toxins, or chemicals that leads to a quantitative or qualitative abnormality; (2) abnormal humoral or cellular control of hematopoiesis; (3) an abnormal or hostile marrow microenvironment; (4) immunologic suppression of hematopoiesis (ie, mediated by antibodies, T cells [or cellularly], or lymphokines); and (5) mutations in genes, causing inherited bone marrow failure syndromes. Identification of these relevant mutations has led to progress in defining the precise functions of the corresponding proteins in normal cells.

Selamat menyambut tahun baru 2011 dan saya berharap masih tidak terlewat untuk mengucapkannya memandangkan kesihatan saya yg makin merosot akhir-akhir ini dan tak dapat nak menumpukan perhatian pada blog.